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1.
Abstract

This study investigated symptoms of exercise-induced muscle damage following a simulated rugby league game. Ten male participants were assessed before, immediately after (0 h), and 24 and 48 h after the simulated game. Perceived muscle soreness was higher at all time points (P=0.001) and creatine kinase values were increased at 24 h following the simulated game (P=0.001). Peak knee extensor torque at 60 deg · s?1 was reduced up to 48 h (P =0.04) but was unchanged at 240 deg · s?1. Similarly, peak knee flexor torque at 60 deg · s?1 was lower than baseline up to 24 h, while at 240 deg · s?1 it was reduced at 24 h only (P=0.045). Correlations between changes in strength loss of the knee extensors and fat mass to fat-free mass ratio reveleaved no significant relationship between variables (P >0.05). In addition, countermovement jump performance was reduced at 0 and 24 h following the simulated game (P=0.008). Our results suggest that symptoms of exercise-induced muscle damage occur up to 48 h following a simulated rugby league match. Coaches should be cognisant of the large increases in muscle soreness and reductions in slow velocity force generation, and should adapt training accordingly in the 48 h period following a game.  相似文献   

2.
Abstract

This study was undertaken to examine the acute effect of interferential current on mechanical pain threshold and isometric peak torque after delayed onset muscle soreness induction in human hamstrings. Forty-one physically active healthy male volunteers aged 18?33 years were randomly assigned to one of two experimental groups: interferential current group (n = 21) or placebo group (n = 20). Both groups performed a bout of 100 isokinetic eccentric maximal voluntary contractions (10 sets of 10 repetitions) at an angular velocity of 1.05 rad · s?1 (60° · s?1) to induce muscle soreness. On the next day, volunteers received either an interferential current or a placebo application. Treatment was applied for 30 minutes (4 kHz frequency; 125 μs pulse duration; 80?150 Hz bursts). Mechanical pain threshold and isometric peak torque were measured at four different time intervals: prior to induction of muscle soreness, immediately following muscle soreness induction, on the next day after muscle soreness induction, and immediately after the interferential current and placebo application. Both groups showed a reduction in isometric torque (P < 0.001) and pain threshold (P < 0.001) after the eccentric exercise. After treatment, only the interferential current group showed a significant increase in pain threshold (P = 0.002) with no changes in isometric torque. The results indicate that interferential current was effective in increasing hamstrings mechanical pain threshold after eccentric exercise, with no effect on isometric peak torque after treatment.  相似文献   

3.
Strenuous physical exercise of the limb muscles commonly results in damage, especially when that exercise is intense, prolonged and includes eccentric contractions. Many factors contribute to exercise-induced muscle injury and the mechanism is likely to differ with the type of exercise. Competitive sports players are highly susceptible to this type of injury. AM3 is an orally administered immunomodulator that reduces the synthesis of proinflammatory cytokines and normalizes defective cellular immune fractions. The ability of AM3 to prevent chronic muscle injury following strenuous exercise characterized by eccentric muscle contraction was evaluated in a double-blind and randomized pilot study. Fourteen professional male volleyball players from the First Division of the Spanish Volleyball League volunteered to take part. The participants were randomized to receive either placebo (n?=?7) or AM3 (n?=?7). The physical characteristics (mean±s) of the placebo group were as follows: age 25.7±2.1 years, body mass 87.2±4.1?kg, height 1.89±0.07?m, maximal oxygen uptake 65.3±4.2?ml?·?kg?1?·?min?1. Those of the AM3 group were as follows: age 26.1±1.9 years, body mass 85.8±6.1?kg, height 1.91±0.07?m, maximal oxygen uptake 64.6±4.5?ml?·?kg?1?·?min?1. All participants were evaluated for biochemical indices of muscle damage, including concentrations of aspartate aminotransferase, alanine aminotransferase, alkaline phosphatase, creatine kinase (CK) and its MB fraction (CK-MB), myoglobin, lactate dehydrogenase, urea, creatinine and γ-glutamyltranspeptidase, both before and 30 days after treatment (over the peak of the competitive season). In the placebo group, competitive exercise (i.e. volleyball) was accompanied by significant increases in creatine kinase (494±51 to 560±53?IU?·?l?1, P?<?0.05) and myoglobin (76.8±2.9 to 83.9±3.1?μg?·?l?1, P?<?0.05); aspartate aminotransferase (30.8±3.0 to 31.1±2.9?IU?·?l?1) and lactate dehydrogenase (380±31 to 376±29?IU?·?l?1) were relatively unchanged after the 30 days maximum effort. AM3 not only inhibited these changes, it led to a decrease from baseline serum concentrations of creatine kinase (503±49 to 316±37?IU?·?l?1, P?<?0.05) and myoglobin (80.1±3.2 to 44.1±2.6?IU?·?l?1, P?<?0.05), as well as aspartate aminotransferase (31.1±3.3 to 26.1±2.7?IU?·?l?1, P?<?0.05) and lactate dehydrogenase (368±34 to 310±3?IU?·?l?1, P?<?0.05). The concentration of CK-MB was also significantly decreased from baseline with AM3 treatment (11.6±1.2 to 5.0±0.7?IU?·?l?1, P?<?0.05), but not with placebo (11.4±1.1 to 10.8±1.4?IU?·?l?1). In conclusion, the use of immunomodulators, such as AM3, by elite sportspersons during competition significantly reduces serum concentrations of proteins associated with muscle damage.  相似文献   

4.
Abstract

Elite badminton requires muscular endurance combined with appropriate maximal and explosive muscle strength. The musculature of the lower extremities is especially important in this context since rapid and forceful movements with the weight of the body are performed repeatedly throughout a match. In the present study, we examined various leg-strength parameters of 35 male elite badminton players who had been performing resistance exercises as part of their physical training for several years. The badminton players were compared with an age-matched reference group, the members of whom were physically active on a recreational basis, and to the same reference group after they had performed resistance training for 14 weeks. Maximal muscle strength of the knee extensor (quadriceps) and flexor muscles (hamstrings) was determined using isokinetic dynamometry. To measure explosive muscle strength, the contractile rate of force development was determined during maximal isometric muscle contractions. In general, the badminton players showed greater maximal muscle strength and contractile rate of force development than the reference group: mean quadriceps peak torque during slow concentric contraction: 3.69 Nm · kg?1, s=0.08 vs. 3.26 Nm · kg?1, s=0.8 (P<0.001); mean hamstring peak torque during slow concentric contraction: 1.86 Nm · kg?1, s=0.04 vs. 1.63 Nm · kg?1, s=0.04 (P<0.001); mean quadriceps rate of force development at 100 ms: 24.4 Nm · s?1·kg?1, s=0.5 vs. 22.1 Nm·s?1 · kg?1, s=0.6 (P<0.05); mean hamstring rate of force development at 100 ms: 11.4 Nm · s?1·kg?1, s=0.3 vs. 8.9 Nm · s?1 · kg?1, s=0.4 (P<0.05). However, after 14 weeks of resistance training the reference group achieved similar isometric and slow concentric muscle strength as the badminton players, although the badminton players still had a higher isometric rate of force development and muscle strength during fast (240° · s?1) quadriceps contractions. Large volumes of concurrent endurance training could have attenuated the long-term development of maximal muscle strength in the badminton players. The badminton players had a higher contractile rate of force development than the reference group before and after resistance training. Greater explosive muscle strength in the badminton players might be a physiological adaptation to their badminton training.  相似文献   

5.
Abstract

The purpose of the present study was to measure and compare peak oxygen uptake and paddling efficiency in recreational and competitive junior male surfers. Eight male recreational surfers (mean age 18 years, s=2; mass 66.8 kg, s=13.0; height 1.75 m, s=0.10) and eight male competitive surfers (mean age 18 years, s=1; mass 68.0 kg, s=11.7; height 1.72 m, s=0.10) performed an incremental paddling test consisting of four 3-min constant load work stages followed by a ramp increase in power output of 20 W · 30 s?1 until exhaustion. The oxygen uptake–power output relationship of the four constant load work stages and peak values obtained during the incremental paddling test were used to calculate paddling efficiency. No differences (P>0.05) were observed between the recreational and competitive surfers for peak oxygen uptake (recreational: 2.52 litres · min?1, s=0.5; competitive: 2.66 litres · min?1, s=0.35) or efficiency (recreational: 24%, s=3; competitive: 21%, s=4). Blood lactate concentration was significantly greater in recreational (2.4 mmol · l?1, s=0.9) than competitive surfers (1.6 mmol · l?1, s=0.5) during submaximal paddling. There were no differences in peak oxygen uptake or paddling efficiency between recreational and competitive surfers suggesting that peak oxygen uptake and efficiency are not sensitive to differences in surfing ability. The increase in blood lactate concentration during submaximal paddling in recreational compared with competitive surfers suggests that other determinants of paddling endurance, such as blood lactate threshold, might be better at distinguishing surfers of differing ability.  相似文献   

6.
Abstract

In this study, we investigated the effect of ingesting carbohydrate alone or carbohydrate with protein on functional and metabolic markers of recovery from a rugby union-specific shuttle running protocol. On three occasions, at least one week apart in a counterbalanced order, nine experienced male rugby union forwards ingested placebo, carbohydrate (1.2 g · kg body mass?1 · h?1) or carbohydrate with protein (0.4 g · kg body mass?1 · h?1) before, during, and after a rugby union-specific protocol. Markers of muscle damage (creatine kinase: before, 258 ± 171 U · L?1 vs. 24 h after, 574 ± 285 U · L?1; myoglobin: pre, 50 ± 18 vs. immediately after, 210 ± 84 nmol · L?1; P < 0.05) and muscle soreness (1, 2, and 3 [maximum soreness = 8] for before, immediately after, and 24 h after exercise, respectively) increased. Leg strength and repeated 6-s cycle sprint mean power were slightly reduced after exercise (93% and 95% of pre-exercise values, respectively; P < 0.05), but were almost fully recovered after 24 h (97% and 99% of pre-exercise values, respectively). There were no differences between trials for any measure. These results indicate that in experienced rugby players, the small degree of muscle damage and reduction in function induced by the exercise protocol were not attenuated by the ingestion of carbohydrate and protein.  相似文献   

7.
Abstract

The aim of this study was to assess the effects of cold-water immersion (cryotherapy) on indices of muscle damage following a bout of prolonged intermittent exercise. Twenty males (mean age 22.3 years, s = 3.3; height 1.80 m, s = 0.05; body mass 83.7 kg, s = 11.9) completed a 90-min intermittent shuttle run previously shown to result in marked muscle damage and soreness. After exercise, participants were randomly assigned to either 10 min cold-water immersion (mean 10°C, s = 0.5) or a non-immersion control group. Ratings of perceived soreness, changes in muscular function and efflux of intracellular proteins were monitored before exercise, during treatment, and at regular intervals up to 7 days post-exercise. Exercise resulted in severe muscle soreness, temporary muscular dysfunction, and elevated serum markers of muscle damage, all peaking within 48 h after exercise. Cryotherapy administered immediately after exercise reduced muscle soreness at 1, 24, and 48 h (P < 0.05). Decrements in isometric maximal voluntary contraction of the knee flexors were reduced after cryotherapy treatment at 24 (mean 12%, s x  = 4) and 48 h (mean 3%, s x  = 3) compared with the control group (mean 21%, s x  = 5 and mean 14%, s x  = 5 respectively; P < 0.05). Exercise-induced increases in serum myoglobin concentration and creatine kinase activity peaked at 1 and 24 h, respectively (P < 0.05). Cryotherapy had no effect on the creatine kinase response, but reduced myoglobin 1 h after exercise (P < 0.05). The results suggest that cold-water immersion immediately after prolonged intermittent shuttle running reduces some indices of exercise-induced muscle damage.  相似文献   

8.
Fatigue represents a reduction in the capability of muscle to generate force. The aim of the present study was to establish the effects of exercise that simulates the work rate of competitive soccer players on the strength of the knee extensors and knee flexors. Thirteen amateur soccer players (age 23.3±3.9 years, height 1.78±0.05?m, body mass 74.8±3.6?kg; mean±s) were tested during the 2000–2001 soccer season. Muscle strength of the quadriceps and hamstrings was measured on an isokinetic dynamometer. A 90?min soccer-specific intermittent exercise protocol, incorporating a 15?min half-time intermission, was developed to provide fatiguing exercise corresponding in work rate to a game of soccer. The exercise protocol, performed on a programmable motorized treadmill, consisted of the different intensities observed during soccer match-play (e.g. walking, jogging, running, sprinting). Muscle strength was assessed before exercise, at half-time and immediately after exercise. A repeated-measures analysis of variance showed significant reductions (P?<0.001) in peak torque for both the quadriceps and hamstrings at all angular velocities (concentric: 1.05, 2.09, 5.23 rad?·?s?1; eccentric: 2.09 rad?·?s?1). The peak torque of the knee extensors (KE) and knee flexors (KF) was greater before exercise [KE: 232±37, 182±34, 129±27, 219±41?N?·?m at 1.05, 2.09 and 5.23 rad?·?s?1 (concentric) and 2.09 rad?·?s?1 (eccentric), respectively; KF: 126±20, 112±19, 101±16, 137±23?N?·?m] than at half-time (KE: 209±45, 177±35, 125±36, 214±43?N?·?m; KF: 114±31, 102±20, 92±15, 125±25?N?·?m) and greater at half-time than after exercise (KE: 196±43, 167±35, 118±24, 204±43?N?·?m; KF: 104±25, 95±21, 87±13, 114±27?N?·?m). For the hamstrings?:?quadriceps ratio, significant changes were found (P?<0.05) for both legs, the ratio being greater before than after exercise. For fast?:?slow speed and left?:?right ratios, no significant changes were found. We conclude that there is a progressive reduction in muscle strength that applies across a range of functional characteristics during exercise that mimics the work rate in soccer.  相似文献   

9.
Abstract

The aim of this study was to examine the effect of menstrual cycle phase on 2000-m rowing ergometry performance. Since high concentrations of oestrogen, indicative of the mid-luteal phase of the menstrual cycle, tend to decrease glycogen utilization and reduce blood lactate concentration, it was predicted that time taken to complete a 2000-m rowing trial would be shorter in the mid-luteal phase. Ten eumenorrhoeic, recreationally trained, female volunteers (mean age 33.0 years, s=7.1) completed 2000-m time trials on a Concept 2 rowing ergometer, in both the mid-follicular and mid-luteal phases of their menstrual cycle. In each phase, a 3-min incremental rowing protocol was used to determine a blood lactate concentration of 4 mmol · l?1 (T lac-4mM) and maximum oxygen consumption (VO2max); a five-stroke maximal test was used to establish maximal power. Order of testing was randomized for menstrual cycle phase. Variables (T lac-4mM, VO2max, maximal power) were correlated with speed in the 2000-m time trials, and the effect of menstrual cycle phase on these variables was examined. A blood lactate concentration of 4 mmol · l?1 occurred at a significantly higher mean exercise intensity (mid-luteal vs. mid-follicular: 169.1 W, s=39.1 vs. 159.0 W, s=38.3; P=0.033), heart rate (179 beats · min?1, s=9 vs. 173 beats · min?1, s=11; P=0.0047), and oxygen consumption (2.64 litres · min?1, s=0.66 vs. 2.42 litres · min?1, s=0.62; P=0.04) in the mid-luteal phase than in the mid-follicular phase. There was no significant difference (P=0.11) in 2000-m time trial speed according to menstrual cycle phase. In conclusion, although T lac-4mM differed due to menstrual cycle phase, 2000-m rowing performance was unaffected. Further research into the effects of menstrual cycle on rowing performance of a longer duration, among a more homogenous group of females, is recommended.  相似文献   

10.
Abstract

The aim of this study was to determine if inducing metabolic alkalosis would alter neuromuscular control after 50 min of standardized submaximal cycling. Eight trained male cyclists (mean age 32 years, s = 7; [Vdot]O2max 62 ml · kg?1 · min?1, s = 8) ingested capsules containing either CaCO3 (placebo) or NaHCO3 (0.3 g · kg?1 body mass) in eight doses over 2 h on two separate occasions, commencing 3 h before exercise. Participants performed three maximal isometric voluntary contractions (MVC) of the knee extensors while determining the central activation ratio by superimposing electrical stimulation both pre-ingestion and post-exercise, followed by a 50-s sustained maximal contraction in which force, EMG amplitude, and muscle fibre conduction velocity were assessed. Plasma pH, blood base excess, and plasma HCO3 were higher (P < 0.01) during the NaHCO3 trial. After cycling, muscle fibre conduction velocity was higher (P < 0.05) during the 50-s sustained maximal contraction with NaHCO3 than with placebo (5.1 m · s?1, s = 0.4 vs. 4.2 m · s?1, s = 0.4) while the EMG amplitude remained the same. Force decline rate was less (P < 0.05) during alkalosis-sustained maximal contraction and no differences were shown in central activation ratio. These data indicate that induced metabolic alkalosis can increase muscle fibre conduction velocity following prolonged submaximal cycling.  相似文献   

11.
Abstract

The aim of this study was to assess the effect of intermittent hypoxia exposure on direct and indirect methods used to evaluate recombinant human erythropoietin (rhEPO) misuse. Sixteen male triathletes were randomly assigned to either the intermittent hypoxia exposure group (experimental group) or the control normoxic group (control group). The members of the experimental group were exposed to simulated altitude (from 4000 to 5500 m) in a hypobaric chamber for 3 h per day, 5 days a week, for 4 weeks. Blood and urine samples were collected before and after the first and the final exposures, and again 2 weeks after the final exposure. While serum EPO significantly increased after the first [from a mean 8.3 IU · l?1 (s = 3.2) to 16.6 IU · l?1 (s = 4.7)] and final exposures [from 4.6 IU · l?1 (s = 1.4) to 24.8 IU · l?1 (s = 9.3)], haemoglobin, percentage of reticulocytes, and soluble transferrin receptor were not elevated. Second-generation ON/OFF models (indirect rhEPO misuse detection) were insensitive to intermittent hypoxia exposure. The distribution of the urinary EPO isoelectric profiles (direct rhEPO misuse detection) was altered after intermittent hypoxia exposure with a slight shift towards more basic isoforms. However, those shifts never resulted in misinterpretation of results. The intermittent hypoxia exposure protocol studied did not produce any false-positive result for indirect or direct detection of rhEPO misuse in spite of the changes in EPO serum concentrations and urinary EPO isoelectric profiles, respectively.  相似文献   

12.
Abstract

The aim of this study was to examine the effects of active versus passive recovery on blood lactate disappearance and subsequent maximal performance in competitive swimmers. Fourteen male swimmers from the University of Virginia swim team (mean age 20.3 years, s = 4.1; stature 1.85 m, s = 2.2; body mass 81.1 kg, s = 5.6) completed a lactate profiling session during which the speed at the lactate threshold (VLT), the speed at 50% of the lactate threshold (VLT.5), and the speed at 150% of the lactate threshold (VLT1.5) were determined. Participants also completed four randomly assigned experimental sessions that consisted of a 200-yard maximal-effort swim followed by 10 min of recovery (passive, VLT.5, VLT, VLT1.5) and a subsequent 200-yard maximal effort swim. All active recovery sessions resulted in greater lactate disappearance than passive recovery (P < 0.0001 for all comparisons), with the greatest lactate disappearance associated with recovery at VLT (P = 0.006 and 0.007 vs. VLT.5 and VLT1.5 respectively) [blood lactate disappearance was 2.1 mmol · l?1 (s = 2.0), 6.0 mmol · l?1 (s = 2.6), 8.5 mmol · l?1 (s = 1.8), and 6.1 mmol · l?1 (s = 2.5) for passive, VLT.5, VLT, and VLT1.5 respectively]. Active recovery at VLT and VLT1.5 resulted in faster performance on time trial 2 than passive recovery (P = 0.005 and 0.03 respectively); however, only active recovery at VLT resulted in improved performance on time trial 2 (TT2) relative to time trial 1 (TT1) [TT2?TT1: passive +1.32 s (s = 0.64), VLT.5+1.01 s (s = 0.53), VLT?1.67 s (s = 0.26), VLT1.5?0.07 s (s = 0.51); P < 0.0001 for VLT). In conclusion, active recovery at the speed associated with the lactate threshold resulted in the greatest lactate disappearance and in improved subsequent performance in all 14 swimmers. Our results suggest that coaches should consider incorporating recovery at the speed at the lactate threshold during competition and perhaps during hard training sessions.  相似文献   

13.
Downhill backwards walking causes repeated, cyclical loading of the muscle–tendon unit. The effect this type of repeated loading has on the mechanical behaviour of the Achilles tendon is presently unknown. This study aimed to investigate the biomechanical response of the Achilles tendon aponeurosis complex following a downhill backwards walking protocol. Twenty active males (age: 22.3 ± 3.0 years; mass: 74.7 ± 5.6 kg; height: 1.8 ± 0.7 m) performed 60 min of downhill (8.5°), backwards walking on a treadmill at ?0.67 m · s?1. Data were collected before, immediately post, and 24-, 48- and 168-h post-downhill backwards walking. Achilles tendon aponeurosis elongation, strain and stiffness were measured using ultrasonography. Muscle force decreased immediately post-downhill backward walking (P = 0.019). There were increases in Achilles tendon aponeurosis stiffness at 24-h post-downhill backward walking (307 ± 179.6 N · mm?1, P = 0.004), and decreases in Achilles tendon aponeurosis strain during maximum voluntary contraction at 24 (3.8 ± 1.7%, P = 0.008) and 48 h (3.9 ± 1.8%, P = 0.002) post. Repeated cyclical loading of downhill backwards walking affects the behaviour of the muscle–tendon unit, most likely by altering muscle compliance, and these changes result in tendon stiffness increases.  相似文献   

14.
Abstract

We investigated the associations of anthropometry, training, and pre-race experience with race time in 93 recreational male ultra-marathoners (mean age 44.6 years, s = 10.0; body mass 74.0 kg, s = 9.0; height 1.77 m, s = 0.06; body mass index 23.4 kg · m?2, s = 2.0) in a 100-km ultra-marathon using bivariate and multivariate analysis. In the bivariate analysis, body mass index (r = 0.24), the sum of eight skinfolds (r = 0.55), percent body fat (r = 0.57), weekly running hours (r = ?0.29), weekly running kilometres (r = ?0.49), running speed during training (r = ?0.50), and personal best time in a marathon (r = 0.72) were associated with race time. Results of the multiple regression analysis revealed an independent and negative association of weekly running kilometres and average speed in training with race time, as well as a significant positive association between the sum of eight skinfold thicknesses and race time. There was a significant positive association between 100-km race time and personal best time in a marathon. We conclude that both training and anthropometry were independently associated with race performance. These characteristics remained relevant even when controlling for personal best time in a marathon.  相似文献   

15.
ABSTRACT

We analysed the time course of recovery of creatine kinase (CK) and countermovement jump (CMJ) parameters after a football match, and correlations between changes in these variables and match time–motion parameters (GPS-accelerometry) in 15 U-19 elite male players. Plasma CK and CMJ height (CMJH), average concentric force (CMJCON) and average eccentric force (CMJECC) were assessed 2 h before and 30 min, 24 h and 48 h post-match. There were substantially higher CK levels 30 min, 24 h and 48 h (ES: 0.43, 0.62, 0.40, respectively), post-match. CMJECC (ES: ?0.38), CMJH (ES: ?0.35) decreased 30 min post, CMJCON (ES: ?0.35), CMJECC (ES: ?0.35) and CMJH (ES: ?1.35) decreased 24 h post, and CMJCON (ES: ?0.41) and CMJH (ES: ?0.53) decreased 48 h post. We found correlations between distance covered at velocities ≤21 km · h?1 and changes in CK at 24 h (r = 0.56) and at 48 h (r = 0.54) and correlations between CK and distance covered >14 km · h?1 (r = 0.50), accelerations (r = 0.48), and decelerations (r = 0.58) at 48 h. Changes in CMJCON 30 min and 24 h post (both r = ?0.68) correlated with impacts >7.1·G. Decelerations >2 m · s?2 correlated with changes CMJCON (r = ?0.49) at 48 h and CMJECC (r = ?0.47) at 30 min. Our results suggest that match GPS-accelerometry parameters may predict muscle damage and changes in components of neuromuscular performance immediately and 24–48 h post-match.  相似文献   

16.
The purpose of this study was to examine the influence of a carbohydrate-rich meal on post-prandial metabolic responses and skeletal muscle glycogen concentration. After an overnight fast, eight male recreational/club endurance runners ingested a carbohydrate (CHO) meal (2.5 g CHO?·?kg?1 body mass) and biopsies were obtained from the vastus lateralis muscle before and 3 h after the meal. Ingestion of the meal resulted in a 10.6?±?2.5% (P?<?0.05) increase in muscle glycogen concentration (pre-meal vs post-meal: 314.0?±?33.9 vs 347.3?±?31.3 mmol?·?kg?1 dry weight). Three hours after ingestion, mean serum insulin concentrations had not returned to pre-feeding values (0 min vs 180 min: 45?±?4 vs 143?±?21 pmol?·?l?1). On a separate occasion, six similar individuals ingested the meal or fasted for a further 3 h during which time expired air samples were collected to estimate the amount of carbohydrate oxidized over the 3 h post-prandial period. It was estimated that about 20% of the carbohydrate consumed was converted into muscle glycogen, and about 12 % was oxidized. We conclude that a meal providing 2.5 g CHO?·?kg?1 body mass can increase muscle glycogen stores 3 h after ingestion. However, an estimated 67% of the carbohydrate ingested was unaccounted for and this may have been stored as liver glycogen and/or still be in the gastrointestinal tract.  相似文献   

17.
Abstract

The current study implemented a two-part design to (1) assess the vitamin D concentration of a large cohort of non-vitamin D supplemented UK-based athletes and 30 age-matched healthy non-athletes and (2) to examine the effects of 5000 IU · day?1 vitamin D3 supplementation for 8-weeks on musculoskeletal performance in a placebo controlled trial. Vitamin D concentration was determined as severely deficient if serum 25(OH)D < 12.5 nmol · l?1, deficient 12.5–30 nmol · l?1 and inadequate 30–50 nmol · l?1. We demonstrate that 62% of the athletes (38/61) and 73% of the controls (22/30) exhibited serum total 25(OH)D < 50 nmol · l?1. Additionally, vitamin D supplementation increased serum total 25(OH)D from baseline (mean ± SD = 29 ± 25 to 103 ± 25 nmol · l?1, P = 0.0028), whereas the placebo showed no significant change (53 ± 29 to 74 ± 24 nmol · l?1, P = 0.12). There was a significant increase in 10 m sprint times (P = 0.008) and vertical-jump (P = 0.008) in the vitamin D group whereas the placebo showed no change (P = 0.587 and P = 0.204 respectively). The current data supports previous findings that athletes living at Northerly latitudes (UK = 53° N) exhibit inadequate vitamin D concentrations (<50 nmol · l?1). Additionally the data suggests that inadequate vitamin D concentration is detrimental to musculoskeletal performance in athletes. Future studies using larger athletic groups are now warranted.  相似文献   

18.
Pacing strategies of elite swimmers have been consistently characterised from the average lap velocities. In the present study, we examined the racing strategies of 200 m world class-level swimmers with regard to their underwater and surface lap components. The finals and semi-finals of the 200 m races at the 2013 World Swimming Championships (Barcelona, Spain) were analysed by an innovative image-processing system (InThePool® 2.0). Free swimming velocities of elite swimmers typically decreased throughout the 200 m race laps (?0.12 m · s–1, 95% CI ?0.11 to ?0.14 m · s–1, P = 0.001, η2 = 0.81), whereas underwater velocities, which were faster than free swimming, were not meaningfully affected by the race progress (0.02 m · s–1, ?0.01 to 0.04 m · s–1, P = 0.01, η2 = 0.04). When swimming underwater, elite swimmers typically travelled less distance (?0.66 m, ?0.83 to ?0.49 m, P = 0.001, η2 = 0.34) from the first to the third turn of the race, although underwater distances were maintained on the backstroke and butterfly races. These strategies allowed swimmers to maintain their average velocity in the last lap despite a decrease in the free swimming velocity. Elite coaches and swimmers are advised to model their racing strategies by considering both underwater and surface race components.  相似文献   

19.
Abstract

Three textiles with increasing compressive surface were compared with non-compressive conventional clothing on physiological and perceptual variables during sub-maximal and maximal running. Fifteen well-trained endurance athletes (mean ± s: age 27.1 ± 4.8 years, [Vdot]O2max 63.7 ± 4.9 ml · min?1 · kg?1) performed four sub-maximal (～70%[Vdot]O2max) and maximal tests with and without different compression stockings, tights, and whole-body compression suits. Arterial lactate concentration, oxygen saturation and partial pressure, pH, oxygen uptake, and ratings of muscle soreness were recorded before, during, and after all tests. In addition, we assessed time to exhaustion. Sub-maximal (P = 0.22) and maximal oxygen uptake (P = 0.26), arterial lactate concentration (P = 0.16; 0.20), pH (P = 0.23; 0.46), oxygen saturation (P = 0.13; 0.26), and oxygen partial pressure (P = 0.09; 0.20) did not differ between the types of clothing (effect sizes = 0.00–0.45). Ratings of perceived exertion (P = 0.10; 0.15), muscle soreness (P = 0.09; 0.10) and time to exhaustion (P = 0.16) were also unaffected by the different clothing (effect sizes = 0.28–0.85). This was the first study to evaluate the effect on endurance performance of different types of compression clothing with increasing amounts of compressive surface. Overall, there were no performance benefits when using the compression garments.  相似文献   

20.
Abstract

The single-stage treadmill walking test of Ebbeling et al. is commonly used to predict maximal oxygen consumption ([Vdot]O2max) from a submaximal effort between 50% and 70% of the participant's age-predicted maximum heart rate. The purpose of this study was to determine if this submaximal test correctly predicts [Vdot]O2max at the low (50% of maximum heart rate) and high (70% of maximum heart rate) ends of the specified heart rate range for males and females aged 18 – 55 years. Each of the 34 participants completed one low-intensity and one high-intensity trial. The two trials resulted in significantly different estimates of [Vdot]O2max (low-intensity trial: mean 40.5 ml · kg?1 · min?1, s = 9.3; high-intensity trial: 47.5 ml · kg?1 · min?1, s = 8.8; P < 0.01). A subset of 22 participants concluded their second trial with a [Vdot]O2max test (mean 47.9 ml · kg?1 · min?1, s = 8.9). The low-intensity trial underestimated (mean difference = ?3.5 ml · kg?1 · min?1; 95% CI = ?6.4 to ?0.6 ml · kg?1 · min?1; P = 0.02) and the high-intensity trial overestimated (mean difference = 3.5 ml · kg?1 · min?1; 95% CI = 1.1 to 6.0 ml · kg?1 · min?1; P = 0.01) the measured [Vdot]O2max. The predictive validity of Ebbeling and colleagues' single-stage submaximal treadmill walking test is diminished when performed at the extremes of the specified heart rate range.  相似文献   

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