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本文给出了时序模型:X_1=ε_1+((?)+η)X_(1-1)一阶平稳的条件,并提出了随机格林函数的概念.  相似文献   
3.
Abstract

In this study, we examined the correlations between selected markers of isometric training intensity and subsequent reductions in resting blood pressure. Thirteen participants performed a discontinuous incremental isometric exercise test to volitional exhaustion at which point mean torque for the final 2-min stage (2min-torquepeak) and peak heart rate peak (HRpeak) were identified. Also, during 4 weeks of training (3 sessions per week, comprising 4 × 2 min bilateral leg isometric exercise at 95% HRpeak), heart rate (HRtrain), torque (Torquetrain), and changes in EMG amplitude (ΔEMGamp) and frequency (ΔEMGfreq) were determined. The markers of training intensity were: Torquetrain relative to the 2min-torquepeak (%2min-torquepeak), EMG relative to EMGpeak (%EMGpeak), HRtrain ΔEMGamp, ΔEMGfreq, and %MVC. Mean systolic (?4.9 mmHg) and arterial blood pressure (?2.7mmHg) reductions correlated with %2min-torquepeak (r = ?0.65, P = 0.02 and r = ?0.59, P = 0.03), ΔEMGamp (r = 0.66, P = 0.01 and r = 0.59, P = 0.03), ΔEMGfreq (r = ?0.67, P = 0.01 and r = ?0.64, P = 0.02), and %EMGpeak (systolic blood pressure only; r = ?0.63, P = 0.02). These markers best reflect the association between isometric training intensity and reduction in resting blood pressure observed after bilateral leg isometric exercise training.  相似文献   
4.
The ideal of personal autonomy enjoys considerable support in educational theory, but close analysis reveals serious problems with its core analytical and psychological components. The core conception of autonomy authorizes individuals to employ their imaginations in troubling and unhealthy ways that clash with sound ideals of moral character. Lucas Swaine argues in this essay that this gives grounds to deny that the core conception of autonomy should be promoted in democratic education. What is more, according to Swaine, young citizens appear to have no right to be educated, in public schools, for the purpose of becoming autonomous individuals of the kind he describes and criticizes in this account.  相似文献   
5.
There is little published data in relation to the effects of caffeine upon cycling performance, speed and power in trained cyclists, especially during cycling of approximately 60 s duration. To address this, eight trained cyclists performed a 1 km time-trial on an electronically braked cycle ergometer under three conditions: after ingestion of 5 mg x kg-1 caffeine, after ingestion of a placebo, or a control condition. The three time-trials were performed in a randomized order and performance time, mean speed, mean power and peak power were determined. Caffeine ingestion resulted in improved performance time (caffeine vs. placebo vs. control: 71.1 +/- 2.0 vs. 73.4 +/- 2.3 vs. 73.3 +/- 2.7 s; P = 0.02; mean +/- s). This change represented a 3.1% (95% confidence interval: 0.7-5.6) improvement compared with the placebo condition. Mean speed was also higher in the caffeine than placebo and control conditions (caffeine vs. placebo vs. control: 50.7 +/- 1.4 vs. 49.1 +/- 1.5 vs. 49.2 +/- 1.7 km x h-1; P = 0.0005). Mean power increased after caffeine ingestion (caffeine vs. placebo vs. control: 523 +/- 43 vs. 505 +/- 46 vs. 504 +/- 38 W; P = 0.007). Peak power also increased from 864 +/- 107 W (placebo) and 830 +/- 87 W (control) to 940 +/- 83 W after caffeine ingestion (P = 0.027). These results provide support for previous research that found improved performance after caffeine ingestion during short-duration high-intensity exercise. The magnitude of the improvements observed in our study could be due to our use of sport-specific ergometry, a tablet form and trained participants.  相似文献   
6.
There is a paucity of research on devices suitable for home-based isometric exercise. Our aim was to compare cardiovascular responses to isometric exercise using novel and established methods. Ten individuals (age 34.0?±?8.5 years, mass 68.2?±?10.4?kg, height 1.72?±?0.09?m; mean?±?s) performed three different isometric exercise protocols with 48?h between each. Each protocol involved four repeated exercise bouts of 2?min at 30% maximum voluntary contraction force using alternate legs (transducer), alternate arms (transducer), or alternate arms (novel device). Systolic blood pressure, diastolic blood pressure, mean arterial blood pressure and heart rate were measured every 30?s. The highest (peak) values during each 2?min bout of exercise were recorded (peak systolic blood pressure, peak diastolic blood pressure, peak mean arterial blood pressure and peak heart rate). At the end of each 2?min exercise bout, the participants rated their perceived discomfort using Borg's CR-10 scale. There was a statistically significant difference in peak systolic blood pressure between isometric arm flexion using the force transducer and the novel device [158.1?±?10.8 vs. 149.1?±?13.9?mmHg (mean?±?s); P = 0.02]. Further analysis showed that peak systolic blood pressure was on average 9?mmHg higher using the force transducer with limits of agreement of –?15.97 to 33.97?mmHg. Analysis of the peak diastolic blood pressure, peak mean arterial blood pressure, peak heart rate and CR-10 data revealed no statistically significant differences between the three protocols. These results suggest that this novel, home-based method elicited similar cardiovascular responses during isometric exercise to those of established laboratory-based methods. However, the lower peak systolic blood pressure using the modified scales warrants further investigation before this method is used widely in the home.  相似文献   
7.
Abstract

The purpose of this study was (a) to assess lactate accumulation during isometric exercise, and to quantify the shifts in accumulation following isometric training; and (b) to relate any training-induced changes in lactate accumulation to reductions in resting blood pressure. Eleven male participants undertook isometric training for a 4-week period using bilateral-leg exercise. Training caused reductions in systolic, diastolic, and mean arterial resting blood pressure (of ?4.9 ± 6.3 mmHg, P = 0.01; ?2.6 ± 3.0 mmHg, P = 0.01; and ?2.6 ± 2.3 mmHg, P = 0.001 respectively; mean ± s). These were accompanied by changes in muscle activity, taken as electromyographic activity to reach a given lactate concentration (from 114 ± 22 to 131 ± 27 mV and from 136 ± 25 to 155 ± 34 mV for 3 and 4 mmol · L?1 respectively. Training intensity expressed relative to peak lactate was correlated with reduced resting systolic and mean arterial blood pressure. Training caused significant shifts in lactate accumulation, and reductions in resting blood pressure are strongly related to training intensity, when expressed relative to pre-training peak lactate. This suggests that higher levels of local muscle anaerobiosis may promote the training-induced reductions in resting blood pressure.  相似文献   
8.
To reduce resting blood pressure, a minimum isometric exercise training (IET) intensity has been suggested, but this is not known for short-term IET programmes. We therefore compared the effects of moderate- and low-intensity IET programmes on resting blood pressure. Forty normotensive participants (22.3 ± 3.4 years; 69.5 ± 15.5 kg; 170.2 ± 8.7 cm) were randomly assigned to groups of differing training intensities [20%EMGpeak (~23%MVC, maximum voluntary contraction, or 30%EMGpeak (~34%MVC)] or control group; 3 weeks of IET at 30%EMGpeak resulted in significant reductions in resting mean arterial pressure (e.g. ?3.9 ± 1.0 mmHg, < 0.001), whereas 20%EMGpeak did not (?2.3 ± 2.9 mmHg; > 0.05). Moreover, after pooling all female versus male participants, IET induced a 6.9-mmHg reduction in systolic blood pressure in female participants, but only a 1.5-mmHg reduction in systolic blood pressure in male participants, although the difference was not significant. An IET intensity between 20%EMGpeak and 30%EMGpeak is sufficient to elicit significant resting blood pressure reductions in a short-term training period (3 weeks). In addition, sexual dimorphism may exist in the magnitude of reductions, but further work is required to confirm this possibility, which could be important in understanding the mechanisms responsible.  相似文献   
9.
Abstract

There is little published data in relation to the effects of caffeine upon cycling performance, speed and power in trained cyclists, especially during cycling of ~60 s duration. To address this, eight trained cyclists performed a 1 km time-trial on an electronically braked cycle ergometer under three conditions: after ingestion of 5 mg · kg?1 caffeine, after ingestion of a placebo, or a control condition. The three time-trials were performed in a randomized order and performance time, mean speed, mean power and peak power were determined. Caffeine ingestion resulted in improved performance time (caffeine vs. placebo vs. control: 71.1 ± 2.0 vs. 73.4 ± 2.3 vs. 73.3 ± 2.7 s; P = 0.02; mean ± s). This change represented a 3.1% (95% confidence interval: 0.7–5.6) improvement compared with the placebo condition. Mean speed was also higher in the caffeine than placebo and control conditions (caffeine vs. placebo vs. control: 50.7 ± 1.4 vs. 49.1 ± 1.5 vs. 49.2 ± 1.7 km · h?1; P = 0.0005). Mean power increased after caffeine ingestion (caffeine vs. placebo vs. control: 523 ± 43 vs. 505 ± 46 vs. 504 ± 38 W; P = 0.007). Peak power also increased from 864 ± 107 W (placebo) and 830 ± 87 W (control) to 940 ± 83 W after caffeine ingestion (P = 0.027). These results provide support for previous research that found improved performance after caffeine ingestion during short-duration high-intensity exercise. The magnitude of the improvements observed in our study could be due to our use of sport-specific ergometry, a tablet form and trained participants.  相似文献   
10.

Assessments of maximal intensity exercise which determine peak power output on friction‐baked cycle ergometers have fallen into two categories: correction procedures which account for changes in momentum of the ergometer's flywheel and optimization procedures which attempt to satisfy muscle force‐velocity relationships. The aim of this study was to compare performance in each procedure and so investigate assumptions which underpin the tests. Nineteen males aged 20.9 ± 0.4 years and 18 females aged 22.2 ± 0.7 years (mean ± S.E.M.), who were fully accustomed to the procedures, participated in a single experimental protocol. After a 5 min warm‐up, the subjects performed four bouts of all‐out exercise on a Monark 814E cycle ergometer against randomly assigned loads. The loads were selected to produce peak pedalling rates in the range 100–200 rev min?1 and each bout lasted 10 s. From the inverse linear relationship between applied load and peak pedalling rate, optimized peak power output (PP opt) and the accompanying pedalling rate (RPM opt) were calculated. One of the bouts used a loading equivalent to 7.5% of body weight and for this bout corrected peak power output (PP corr) and its corresponding pedalling rate (RPM corr) were calculated. The PP opt was less than PP corr in the males (915 ± 35 vs 1005 ± 32 W) and females (673 ± 33 vs 777 ± 39 W) (both P < 0.001). Similarly, RPM opt was less than RPM corr (111 ± 1 vs 128 ± 2 rev min?1 and 101 ± 1 vs 111 ± 2 rev min?1 in the males and females, respectively; P < 0.001). The results demonstrate that optimization and correction procedures produce different values of performance. These differences are probably attributable to the mechanical principles which underpin the tests.  相似文献   
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