排序方式： 共有92条查询结果，搜索用时 18 毫秒
81.
足球运动员补充糖和/或肌酸对运动后血乳酸的影响 总被引：9，自引：1，他引：8
8名男性足球运动员,按正交表L4(23)进行实验设计,进行关于糖、肌酸补充对足球运动员体能影响的效果观察实验.实验结果表明:补糖可以显著降低赛后血乳酸水平,补肌酸对赛后血乳酸水平无显著性影响,二者无显著性交互作用;补糖能显著性升高力竭运动后血乳酸值,补肌酸对力竭运动后血乳酸值无显著性影响,二者无显著性交互作用.结果提示补充糖有助于提高运动员无氧能力,有利于足球运动员提高赛场竞技能力. 相似文献
82.
中学数学教学培养学生创新能力的方法 总被引：1，自引：0，他引：1
结合中学数学教学的实践谈培养学生创新能力的途径和方法。 相似文献
83.
Enzymology is a diagnostic indicator for myocardial infarction and diabetes in hypertension patients. Therefore the selection
of methods for measurement of cardiac enzyme, Aspartate transaminase (AST), Creatine kinase(CK), and isoenzyme of creatine
kinase (MB form), determine the effectiveness of antihypertension drug would provide the physician with diagnostic and prognostic
clinical evidence. 相似文献
84.
补充丙酮酸肌酸、肌酸和肉碱对运动员身体成分及运动能力的影响 总被引：1，自引：0，他引：1
目的:观察运动员在系统训练过程中服用丙酮酸肌酸、肌酸和肉碱时身体成分及运动能力的不同影响.方法:36名国家青年篮球队退动员,随机分为安慰剂组、丙酮酸肌酸组、肌酸组和肉碱组.正常训练同时持续6周服用营养补剂和安慰剂,实验前、后进行身体成分测试和运动能力测试.结果:在系统训练中使用丙酮酸肌酸可以明显减少运动员的脂肪,降低体脂百分数;能够明显增加运动员肌肉重量和去脂体重;明显提高运动员运动能力.男运动员在系统训练中使用肌酸和丙酮酸肌酸,可以减少身体脂肪,但使用丙酮酸肌酸减少脂肪的幅度更大;同时,使用丙酮酸肌酸还能够明显增加肌肉重量;在系统训练中使用丙酮酸肌酸比肌酸更能提高运动能力且没有副反应.女运动员在训练中使用肉碱和丙酮酸肌酸均能减少身体脂肪、提高运动能力,丙酮酸肌酸的效果更明显.结论:在系统训练中使用丙酮酸肌酸比肌酸和肉碱更能减少运动员身体脂肪、增加肌肉重量,提高运动能力. 相似文献
85.
艾灸对运动员运动时机体血清酶活性的影响 总被引：13，自引：0，他引：13
通过对运动员进行穴位施灸处理，并以服用西洋参组作为对照，观察定量运动时机体血清肌酸激酶(CK)活性的变化和血清超氧化物歧化酶（SOD）活性的变化。经过2个月的施灸处理，结果表明，艾灸可以明显降低运动员运动时机体血清肌酸激酶(CK)的活性，同时使SOD活性保持相对稳定。提示，灸法可以提高机体对运动的适应能力，减轻运动性疲劳。 相似文献
86.
90～100日龄 Wisten雄性大 21只,随机分成 3 组,并在同样条件下饲养,其中一组在安静条件下处死,另一组在力竭性游泳2小时后处死,第三组在力竭性游泳后立即处死.然后检测它们的血乳酸、血肌酐、血尿酸水平,结果显示力竭性运动后血乳酸增多以及肾血流量和肾过滤率下降是血尿酸水平上升的主要原因。 相似文献
87.
摔跤运动员模拟比赛后疲劳和恢复的生化研究 总被引：12，自引：0，他引：12
通过对北京体育大学10名摔跤运动员摔跤模拟比赛后血、尿一系列生化指标的实验分析，发现摔跤是以糖酵解系统为主、无氧有氧代谢供能的运动项目。在模拟比赛后，血乳酸明显升高，但1h后消除迅速；肌内嘌呤核苷酸分解激活，血氨和尿酸生成增多，血氨和血尿酸与血乳酸指标一样，可作为运动强度和机能恢复的指标。摔跤比赛降低肾功能，赛后60min明显改善，摔跤比赛后至少24h内，机体处于蛋白质净降解状态，约2d后完全恢复。摔跤比赛对运动肌细胞有一定的损伤作用，对结缔组织损伤不明显。 相似文献
88.
《European Journal of Sport Science》2013,13(1):27-33
Abstract In this study, we examined indirect markers of muscle damage and muscle soreness following a 50-km cross-country ski race completed in 2 h and 57 min to 5 h and 9 min by 11 moderately trained male university students. Maximal strength of the knee extensors, several blood markers of muscle damage and inflammation, and muscle soreness (visual analog scale: 0 = “no pain”, 50 mm = “unbearably painful”) were measured one day before, immediately after, and 24, 48, 72, and 144 h after the race. Changes in the measures over time were analysed using one-way repeated-measures analysis of variance and a Fisher's post-hoc test. Maximal strength of the knee extensors decreased significantly (P<0.05) immediately after the race (mean ?27%, s=6), but returned to pre-exercise values within 24 h of the race. All blood markers increased significantly (P<0.05) following the race, peaking either immediately (lactate dehydrogenase: 253.7 IU · l?1, s=13.3; myoglobin: 476.4 ng · ml?1, s=85.5) or 24 h after the race (creatine kinase: 848.0 IU · l?1, s=151.9; glumatic oxaloacetic transaminase: 44.3 IU · l?1, s=4.2; aldolase: 10.0 IU · l?1, s=1.3; C-reactive protein: 0.36 IU · l?1, s=0.08). Muscle soreness developed in the leg, arm, shoulder, back, and abdomen muscles immediately after the race (10–30 mm), but decreased after 24 h (<15 mm), and disappeared 48 h after the race. These results suggest that muscle damage induced by a 50-km cross-country ski race is mild and recovery from the race does not take long. 相似文献
89.
Alfredo Córdova J Francisco Martin Eduardo Reyes Melchor Alvarez-Mon 《Journal of sports sciences》2013,31(9):827-833
Strenuous physical exercise of the limb muscles commonly results in damage, especially when that exercise is intense, prolonged and includes eccentric contractions. Many factors contribute to exercise-induced muscle injury and the mechanism is likely to differ with the type of exercise. Competitive sports players are highly susceptible to this type of injury. AM3 is an orally administered immunomodulator that reduces the synthesis of proinflammatory cytokines and normalizes defective cellular immune fractions. The ability of AM3 to prevent chronic muscle injury following strenuous exercise characterized by eccentric muscle contraction was evaluated in a double-blind and randomized pilot study. Fourteen professional male volleyball players from the First Division of the Spanish Volleyball League volunteered to take part. The participants were randomized to receive either placebo (n?=?7) or AM3 (n?=?7). The physical characteristics (mean±s) of the placebo group were as follows: age 25.7±2.1 years, body mass 87.2±4.1?kg, height 1.89±0.07?m, maximal oxygen uptake 65.3±4.2?ml?·?kg?1?·?min?1. Those of the AM3 group were as follows: age 26.1±1.9 years, body mass 85.8±6.1?kg, height 1.91±0.07?m, maximal oxygen uptake 64.6±4.5?ml?·?kg?1?·?min?1. All participants were evaluated for biochemical indices of muscle damage, including concentrations of aspartate aminotransferase, alanine aminotransferase, alkaline phosphatase, creatine kinase (CK) and its MB fraction (CK-MB), myoglobin, lactate dehydrogenase, urea, creatinine and γ-glutamyltranspeptidase, both before and 30 days after treatment (over the peak of the competitive season). In the placebo group, competitive exercise (i.e. volleyball) was accompanied by significant increases in creatine kinase (494±51 to 560±53?IU?·?l?1, P?<?0.05) and myoglobin (76.8±2.9 to 83.9±3.1?μg?·?l?1, P?<?0.05); aspartate aminotransferase (30.8±3.0 to 31.1±2.9?IU?·?l?1) and lactate dehydrogenase (380±31 to 376±29?IU?·?l?1) were relatively unchanged after the 30 days maximum effort. AM3 not only inhibited these changes, it led to a decrease from baseline serum concentrations of creatine kinase (503±49 to 316±37?IU?·?l?1, P?<?0.05) and myoglobin (80.1±3.2 to 44.1±2.6?IU?·?l?1, P?<?0.05), as well as aspartate aminotransferase (31.1±3.3 to 26.1±2.7?IU?·?l?1, P?<?0.05) and lactate dehydrogenase (368±34 to 310±3?IU?·?l?1, P?<?0.05). The concentration of CK-MB was also significantly decreased from baseline with AM3 treatment (11.6±1.2 to 5.0±0.7?IU?·?l?1, P?<?0.05), but not with placebo (11.4±1.1 to 10.8±1.4?IU?·?l?1). In conclusion, the use of immunomodulators, such as AM3, by elite sportspersons during competition significantly reduces serum concentrations of proteins associated with muscle damage. 相似文献
90.
Craig Sale Roger C. Harris James Florance Alain Kumps Robertine Sanvura Jacques R. Poortmans 《Journal of sports sciences》2013,31(7):759-766
Abstract In this study, we examined the effect of two creatine monohydrate supplementation regimes on 24-h urinary creatine and methylamine excretion. Nine male participants completed two trials, separated by 6 weeks. Participants ingested 4 × 5 g · day?1 creatine monohydrate for 5 days in one trial and 20 × 1 g · day?1 for 5 days in the other. We collected 24-h urine samples on 2 baseline days (days 1–2), during 5 days of supplementation (days 3–7), and for 2 days post-supplementation (days 8–9). Urine was assayed for creatine using high-performance liquid chromatography and methylamine using gas chromatography. Less creatine was excreted following the 20 × 1 g · day?1 regime (49.25 ± 10.53 g) than the 4 × 5 g · day?1 regime (62.32 ± 9.36 g) (mean ± s; P < 0.05). Mean total excretion of methylamine (n = 6) over days 3–7 was 8.61 ± 7.58 mg and 24.81 ± 25.76 mg on the 20 × 1 g · day?1 and 4 × 5 g · day?1 regimes, respectively (P < 0.05). The lower excretion of creatine using 20 × 1 g · day?1 doses suggests a greater retention in the body and most probably in the muscle. Lower and more frequent doses of creatine monohydrate appear to further attenuate formation of methylamine. 相似文献