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1.
The aim of this study was to monitor longitudinal changes in young people's submaximal oxygen uptake (VO 2 ) responses during horizontal treadmill running at 8 km h -1 . The 236 participants (118 boys, 118 girls) were aged 11.2 +/- 0.4 years (mean +/- s) at the onset of the study. Submaximal VO 2 , peak VO 2 and anthropometry were recorded annually for three consecutive years. The data were analysed using multi-level regression modelling within a multiplicative, allometric framework. The initial model examined sex, age and maturity-related changes in submaximal VO 2 relative to body mass as the sole anthropometric covariate. Our results demonstrate that the conventional ratio standard ml kg -1 min -1 does not adequately describe the true relationship between body mass and submaximal VO 2 during this period of growth. The effects of maturity and age were non-significant, but girls consumed significantly less VO 2 than boys running at 8 km h -1 . In subsequent models, stature was shown to be a significant explanatory variable, but this effect became non-significant when the sum of two skinfolds was added. Thus, within this population, submaximal VO 2 responses were explained predominantly by changes in body mass and skinfold thicknesses, with no additional maturity-related increments. When differences in body mass and skinfolds were controlled for, there was still a difference between the sexes in submaximal VO 2 , with girls becoming increasingly more economical with age.  相似文献   

2.
Longitudinal changes in submaximal oxygen uptake in 11- to 13-year-olds   总被引:3,自引:0,他引:3  
The aim of this study was to monitor longitudinal changes in young people's submaximal oxygen uptake (VO2) responses during horizontal treadmill running at 8 km x h(-1). The 236 participants (118 boys, 118 girls) were aged 11.2+/-0.4 years (mean +/- s) at the onset of the study. Submaximal VO2, peak VO2 and anthropometry were recorded annually for three consecutive years. The data were analysed using multi-level regression modelling within a multiplicative, allometric framework. The initial model examined sex, age and maturity-related changes in submaximal VO2 relative to body mass as the sole anthropometric covariate. Our results demonstrate that the conventional ratio standard ml x kg(-1) x min(-1) does not adequately describe the true relationship between body mass and submaximal VO2 during this period of growth. The effects of maturity and age were non-significant, but girls consumed significantly less VO2 than boys running at 8 km x h(-1). In subsequent models, stature was shown to be a significant explanatory variable, but this effect became non-significant when the sum of two skinfolds was added. Thus, within this population, submaximal VO2 responses were explained predominantly by changes in body mass and skinfold thicknesses, with no additional maturity-related increments. When differences in body mass and skinfolds were controlled for, there was still a difference between the sexes in submaximal VO2, with girls becoming increasingly more economical with age.  相似文献   

3.
The aims of this study were: (1) to identify the exercise intensity that corresponds to the maximal lactate steady state in adolescent endurance-trained runners; (2) to identify any differences between the sexes; and (3) to compare the maximal lactate steady state with commonly cited fixed blood lactate reference parameters. Sixteen boys and nine girls volunteered to participate in the study. They were first tested using a stepwise incremental treadmill protocol to establish the blood lactate profile and peak oxygen uptake (VO2). Running speeds corresponding to fixed whole blood lactate concentrations of 2.0, 2.5 and 4.0 mmol x l(-1) were calculated using linear interpolation. The maximal lactate steady state was determined from four separate 20-min constant-speed treadmill runs. The maximal lactate steady state was defined as the fastest running speed, to the nearest 0.5 km x h(-1), where the change in blood lactate concentration between 10 and 20 min was < 0.5 mmol x l(-1). Although the boys had to run faster than the girls to elicit the maximal lactate steady state (15.7 vs 14.3 km x h(-1), P < 0.01), once the data were expressed relative to percent peak VO2 (85 and 85%, respectively) and percent peak heart rate (92 and 94%, respectively), there were no differences between the sexes (P > 0.05). The running speed and percent peak VO2 at the maximal lactate steady state were not different to those corresponding to the fixed blood lactate concentrations of 2.0 and 2.5 mmol x l(-1) (P > 0.05), but were both lower than those at the 4.0 mmol x l(-1) concentration (P < 0.05). In conclusion, the maximal lactate steady state corresponded to a similar relative exercise intensity as that reported in adult athletes. The running speed, percent peak VO2 and percent peak heart rate at the maximal lactate steady state are approximated by the fixed blood lactate concentration of 2.5 mmol x l(-1) measured during an incremental treadmill test in boys and girls.  相似文献   

4.
The purpose of the present study was to assess fitness and running performance in a group of recreational runners (men, n = 18; women, n = 13). 'Fitness' was determined on the basis of their physiological and metabolic responses during maximal and submaximal exercise. There were strong correlations between VO2 max and treadmill running speeds equivalent to blood lactate concentrations of 2 mmol l-1 (V-2 mM) or 4 mmol l-1 (V-4 mM), 'relative running economy' and 5 km times (r = -0.84), but modest and non-significant correlations between muscle fibre composition and running performance. The results of the submaximal exercise tests suggested that the female runners were as well trained as the male runners. However, the men still recorded faster 5 km times (19.20 +/- 1.97 min vs 20.97 +/- 1.70 min; P less than 0.05). Therefore the of the present study suggest that the faster performance times recorded by the men were best explained by their higher VO2 max values, rather than their training status per se.  相似文献   

5.
The aim of this study was to assess the sensitivity of the lactate minimum speed test to changes in endurance fitness resulting from a 6 week training intervention. Sixteen participants (mean +/- s: age 23+/-4 years; body mass 69.7+/-9.1 kg) completed 6 weeks of endurance training. Another eight participants (age 23+/-4 years; body mass 72.7+/-12.5 kg) acted as non-training controls. Before and after the training intervention, all participants completed: (1) a standard multi-stage treadmill test for the assessment of VO2max, running speed at the lactate threshold and running speed at a reference blood lactate concentration of 3 mmol x l(-1); and (2) the lactate minimum speed test, which involved two supramaximal exercise bouts and an 8 min walking recovery period to increase blood lactate concentration before the completion of an incremental treadmill test. Additionally, a subgroup of eight participants from the training intervention completed a series of constant-speed runs for determination of running speed at the maximal lactate steady state. The test protocols were identical before and after the 6 week intervention. The control group showed no significant changes in VO2max, running speed at the lactate threshold, running speed at a blood lactate concentration of 3 mmol x l(-1) or the lactate minimum speed. In the training group, there was a significant increase in VO2max (from 47.9+/-8.4 to 52.2+/-2.7 ml x kg(-1) x min(-1)), running speed at the maximal lactate steady state (from 13.3+/-1.7 to 13.9+/-1.6 km x h(-1)), running speed at the lactate threshold (from 11.2+/-1.8 to 11.9+/-1.8 km x h(-1)) and running speed at a blood lactate concentration of 3 mmol x l(-1) (from 12.5+/-2.2 to 13.2+/-2.1 km x h(-1)) (all P < 0.05). Despite these clear improvements in aerobic fitness, there was no significant difference in lactate minimum speed after the training intervention (from 11.0+/-0.7 to 10.9+/-1.7 km x h(-1)). The results demonstrate that the lactate minimum speed, when assessed using the same exercise protocol before and after 6 weeks of aerobic exercise training, is not sensitive to changes in endurance capacity.  相似文献   

6.
The aim of this study was to assess the sensitivity of the lactate minimum speed test to changes in endurance fitness resulting from a 6 week training intervention. Sixteen participants (mean +/- s :age 23 +/- 4 years;body mass 69.7 +/- 9.1 kg) completed 6 weeks of endurance training. Another eight participants (age 23 +/- 4 years; body mass 72.7 +/-12.5 kg) acted as non-training controls. Before and after the training intervention, all participants completed: (1) a standard multi-stage treadmill test for the assessment of VO 2max , running speed at the lactate threshold and running speed at a reference blood lactate concentration of 3 mmol.l -1 ; and (2) the lactate minimum speed test, which involved two supramaximal exercise bouts and an 8 min walking recovery period to increase blood lactate concentration before the completion of an incremental treadmill test. Additionally, a subgroup of eight participants from the training intervention completed a series of constant-speed runs for determination of running speed at the maximal lactate steady state. The test protocols were identical before and after the 6 week intervention. The control group showed no significant changes in VO 2max , running speed at the lactate threshold, running speed at a blood lactate concentration of 3 mmol.l -1 or the lactate minimum speed.In the training group, there was a significant increase in VO 2max (from 47.9 +/- 8.4 to 52.2 +/- 2.7 ml.kg -1 .min -1 ), running speed at the maximal lactate steady state (from 13.3 +/- 1.7 to 13.9 +/- 1.6 km.h -1 ), running speed at the lactate threshold (from 11.2 +/- 1.8 to 11.9 +/- 1.8 km.h -1 ) and running speed at a blood lactate concentration of 3 mmol.l -1 (from 12.5 +/- 2.2 to 13.2 +/- 2.1 km.h -1 ) (all P ? 0.05). Despite these clear improvements in aerobic fitness, there was no significant difference in lactate minimum speed after the training intervention (from 11.0 +/- 0.7 to 10.9 +/- 1.7 km.h -1 ). The results demonstrate that the lactate minimum speed,when assessed using the same exercise protocol before and after 6 weeks of aerobic exercise training, is not sensitive to changes in endurance capacity.  相似文献   

7.
The literature suggests that the oxygen uptake (VO2) response to the onset of moderate-intensity exercise may be both mature from childhood and independent of sex. Yet the cardiorespiratory response to exercise and the metabolic profile of the muscle appear to change with growth and development and to differ between the sexes. The aim of this study was to investigate further changes in the VO2 kinetic response with age and sex. Participants completed a series of no less than four step change transitions, from unloaded pedalling to a constant work rate corresponding to 80% of their previously determined ventilatory threshold. Each participant's breath-by-breath responses were interpolated to 1 s intervals, time aligned and then averaged. A single exponential model that included a time delay was used to analyse the averaged response following phase 1 (15 s). Participants with parameter confidence intervals more than +/- 5 s were removed from the sample; the results for the remaining 13 men and 12 women (age 19-26 years), 12 boys and 11 girls (age 11-12 years) were used for statistical analysis. Children had a significantly shorter time constant than adults, both for males (19.0+/-2.0 and 27.9+/-8.6 s respectively; P<0.01) and females (21.0+/-5.5 and 26.0+/-4.5 s respectively; P<0.05). There were no significant differences in the time constant between the sexes for either adults or children (P>0.05). A significant relationship between the time constant and peak VO2 was found only in adult males (P<0.05). A shorter time constant in children may reflect an enhanced potential for oxidative metabolism.  相似文献   

8.
The aim of this study was to assess the responses of blood lactate and pyruvate during the lactate minimum speed test. Ten participants (5 males, 5 females; mean +/- s: age 27.1+/-6.7 years, VO2max 52.0+/-7.9 ml x kg(-1) x min(-1)) completed: (1) the lactate minimum speed test, which involved supramaximal sprint exercise to invoke a metabolic acidosis before the completion of an incremental treadmill test (this results in a 'U-shaped' blood lactate profile with the lactate minimum speed being defined as the minimum point on the curve); (2) a standard incremental exercise test without prior sprint exercise for determination of the lactate threshold; and (3) the sprint exercise followed by a passive recovery. The lactate minimum speed (12.0+/-1.4 km x h(-1)) was significantly slower than running speed at the lactate threshold (12.4+/-1.7 km x h(-1)) (P < 0.05), but there were no significant differences in VO2, heart rate or blood lactate concentration between the lactate minimum speed and running speed at the lactate threshold. During the standard incremental test, blood lactate and the lactate-to-pyruvate ratio increased above baseline values at the same time, with pyruvate increasing above baseline at a higher running speed. The rate of lactate, but not pyruvate, disappearance was increased during exercising recovery (early stages of the lactate minimum speed incremental test) compared with passive recovery. This caused the lactate-to-pyruvate ratio to fall during the early stages of the lactate minimum speed test, to reach a minimum point at a running speed that coincided with the lactate minimum speed and that was similar to the point at which the lactate-to-pyruvate ratio increased above baseline in the standard incremental test. Although these results suggest that the mechanism for blood lactate accumulation at the lactate minimum speed and the lactate threshold may be the same, disruption to normal submaximal exercise metabolism as a result of the preceding sprint exercise, including a three- to five-fold elevation of plasma pyruvate concentration, makes it difficult to interpret the blood lactate response to the lactate minimum speed test. Caution should be exercised in the use of this test for the assessment of endurance capacity.  相似文献   

9.
The aim of this study was to assess the responses of blood lactate and pyruvate during the lactate minimum speed test. Ten participants (5 males, 5 females; mean +/- s: age 27.1 +/- 6.7 years, VO 2max 52.0 +/- 7.9 ml kg -1 min -1 ) completed: (1) the lactate minimum speed test, which involved supramaximal sprint exercise to invoke a metabolic acidosis before the completion of an incremental treadmill test (this results in a ‘U-shaped’ blood lactate profile with the lactate minimum speed being defined as the minimum point on the curve); (2) a standard incremental exercise test without prior sprint exercise for determination of the lactate threshold; and (3) the sprint exercise followed by a passive recovery. The lactate minimum speed (12.0 +/- 1.4 km h -1 ) was significantly slower than running speed at the lactate threshold (12.4 +/- 1.7 km h -1 ) (P < 0.05), but there were no significant differences in VO 2 , heart rate or blood lactate concentration between the lactate minimum speed and running speed at the lactate threshold. During the standard incremental test, blood lactate and the lactate-topyruvate ratio increased above baseline values at the same time, with pyruvate increasing above baseline at a higher running speed. The rate of lactate, but not pyruvate, disappearance was increased during exercising recovery (early stages of the lactate minimum speed incremental test) compared with passive recovery. This caused the lactate-to-pyruvate ratio to fall during the early stages of the lactate minimum speed test, to reach a minimum point at a running speed that coincided with the lactate minimum speed and that was similar to the point at which the lactate-to-pyruvate ratio increased above baseline in the standard incremental test. Although these results suggest that the mechanism for blood lactate accumulation at the lactate minimum speed and the lactate threshold may be the same, disruption to normal submaximal exercise metabolism as a result of the preceding sprint exercise, including a three- to five-fold elevation of plasma pyruvate concentration, makes it difficult to interpret the blood lactate response to the lactate minimum speed test. Caution should be exercised in the use of this test for the assessment of endurance capacity.  相似文献   

10.
目的通过实验研究建立青少年“阳光长跑”个体适宜强度(无氧阈心率)回归方程,以改变目前全国青少年“阳光长跑”中强度随意的现状。方法以我国X市3所(A、B、C)中学的高中生为样本,按随机抽样原则从中抽取研究对象,分2个阶段进行研究。①从3所中学高中各年级中抽取男、女生共593名,通过实验测试获得基础数据,以文献资料法和专家法筛选出的指标为自变量、无氧阈心率为因变量,建立个体无氧阈心率回归方程。②来自3所中学高二年级的学生(共204名),分别以3种模式[按照无氧阈心率强度(基于所建立的个体无氧阈心率回归方程)监控、不按强度但严格监控、不监控自主式]完成“阳光长跑”,12周后,比较实验前后3所中学高中生最大摄氧量(VO2max)、基础心率指标的变化情况。结果3所中学高中生按不同模式完成长跑后,其基础心率均未出现降低现象(P均大于0.05);A中学男、女生VO2max均出现不同程度增长现象(t=2.804,P<0.01;t=2.831,P<0.01);B中学女生VO2max出现一定程度增长现象(t=2.337,P<0.05),男生则未出现增长现象(t=0.574,P>0.05);C中学男、女生VO2max均未出现增长现象(P均大于0.05)。结论①所建立的高中生个体无氧阈心率回归方程可用于指导该群体在长跑中的适宜强度调节;②通过实证研究提出针对性建议,有助于“阳光长跑”的可持续性开展。  相似文献   

11.
The purpose of this study was to develop a submaximal, 1.5-mile endurance test for college-aged students using walking, jogging, or running exercise. College students (N = 101: 52 men, 47 women), ages 18-26years, successfully completed the 1.5-mile test twice, and a maximal graded exercise test. Participants were instructed to achieve a "somewhat hard" exercise intensity (rating of perceived exertion = 13) and maintain a steady pace throughout each 1.5-mile test. Multiple linear regression generated the following prediction equation: VO2 max = 65.404 + 7.707 x gender (1 = male; 0 =female) - 0.159 x body mass (kg) - 0.843 x elapsed exercise time (min; walking, jogging orrunning). This equation shows acceptable validity (R = .86, SEE = 3.37 ml x kg(-1) min(-1)) similar to the accuracy of comparable field tests, and reliability (ICC = .93) is also comparable to similar models. The statistical shrinkage is minimal (R(press) = 0.85, SEE(press) = 3.51 ml x kg(-) x min(-1)); hence, it should provide comparable results when applied to other similar samples. A regression model (R =.90, and SEE = 2.87 ml x kg(-1) min(-1)) including exercise heart rate was also developed: VO2 max = 100.162 +/- 7.301 x gender(1 = male; 0 =female) - 0.164 x body mass (kg) - 1.273 x elapsed exercise time -0.156 x exercise heart rate, for those who have access to electronic heart rate monitors. This submaximal 1.5-mile test accurately predicts maximal oxygen uptake (VO2max) without measuring heart rate and is similar to the 1.5-mile run in that it allowsfor mass testing and requires only a flat, measured distance and a stopwatch. Further, it can accommodate a wide range of fitness levels (from walkers to runners).  相似文献   

12.
The purpose of this study was to compare cardiovascular fitness between obese and nonobese children. Based on body mass index, 118 were classified as obese (boys [OB] = 62, girls [OG] = 56), while 421 were nonobese (boys [NOB] = 196, girls [NOG] = 225). Cardiovascular fitness was determined by a 1-mile [1.6 km] run/walk (MRW) and estimated peak oxygen uptake (VO2peak) and analyzed using two-way analyses of variance (Gender x Obese/Nonobese). MRW times were significantly faster (p < .05) for the NOB (10 min 34 s) compared to the OB (13 min 8 s) and the NOG (13 min 15 s.) compared to the OG (14 min 44 s.). Predicted VO2peak values (mL x kg(-1) x min(-1)) were significantly higher (p < .05) for the NOB (48.29) compared to the OB (41.56) and the NOG (45.99) compared to the OG (42.13). MRW was compared between obese and nonobese participants on the President's Challenge (2005), the National Children and Youth Fitness Study, and FITNESSGRAM HFZ standards. The nonobese boys and girls scored higher on all three, exhibiting better cardiovascular fitness as compared to obese counterparts.  相似文献   

13.
Oxygen uptake (VO2) during treadmill exercise is directly related to the speed and grade, as well as the participant's body weight. To determine whether body composition also affects VO2 (ml.kg-1.min-1) during exercise, we studied 14 male body builders (M weight = 99 kg, SD = 7; M height = 180 cm, SD = 8; M body fat = 8%, SD = 3; M fat free mass = 91 kg, SD = 7) and 14 weight-matched men (M weight = 99 kg, SD = 9; M height = 179 cm, SD = 5; M body fat = 24%, SD = 5; M fat free mass = 73 kg, SD = 9). Percentage of body fat, t(13) = 8.185, p < .0001, and fat free mass, t(13) = 5.723, p < .0001, were significantly different between groups. VO2 was measured by respiratory gas analysis at rest and during three different submaximal workrates while walking on the treadmill without using the handrails for support. VO2 was significantly greater for the lean, highly muscular men at rest: 5.6 +/- 1 vs. 4.0 +/- 1 ml.kg-1.min-1, F(1, 26) = 21.185, p < .001; Stage 1: 1.7 mph/10%, 18.5 +/- 2 vs. 16.1 +/- 2 ml.kg-1.min-1, F(1, 26) = 6.002, p < .05; Stage 2: 2.5 mph/12%, 26.6 +/- 3 vs. 23.1 +/- 2 ml.kg-1.min-1, F(1, 26) = 7.991, p < .01; and Stage 3:3.4 mph/14%, 39.3 +/- 5 vs. 33.5 +/- 5 ml.kg-1.min-1, F(1, 26) = 7.682, p < .01, body builders versus weight-matched men, respectively. However, net VO2 (i.e., exercise VO2 - rest VO2) was not significantly different between the two groups at any of the matched exercise stages. The findings from this study indicate that VO2 during weight-bearing exercise performed at the same submaximal workrate is higher for male body builders compared to that measured in weight-matched men and that which is predicted by standard equations. These observed differences in exercise VO2 appear to be due to the higher resting VO2 in highly muscular participants.  相似文献   

14.
Maximal oxygen uptake VO(2max)) is considered the optimal method to assess aerobic fitness. The measurement of VO(2max), however, requires special equipment and training. Maximal exercise testing with determination of maximal power output offers a more simple approach. This study explores the relationship between [Vdot]O(2max) and maximal power output in 247 children (139 boys and 108 girls) aged 7.9-11.1 years. Maximal oxygen uptake was measured by indirect calorimetry during a maximal ergometer exercise test with an initial workload of 30 W and 15 W x min(-1) increments. Maximal power output was also measured. A sample (n = 124) was used to calculate reference equations, which were then validated using another sample (n = 123). The linear reference equation for both sexes combined was: VO(2max) (ml x min(-1)) = 96 + 10.6 x maximal power + 3.5 . body mass. Using this reference equation, estimated VO(2max) per unit of body mass (ml x min(-1) x kg(-1)) calculated from maximal power correlated closely with the direct measurement of VO(2max) (r = 0.91, P <0.001). Bland-Altman analysis gave a mean limits of agreement of 0.2+/-2.9 (ml x min(-1) x kg(-1)) (1 s). Our results suggest that maximal power output serves as a good surrogate measurement for VO(2max) in population studies of children aged 8-11 years.  相似文献   

15.
The aims of this study were: (1) to identify the exercise intensity that corresponds to the maximal lactate steady state in adolescent endurance-trained runners; (2) to identify any differences between the sexes; and (3) to compare the maximal lactate steady state with commonly cited fixed blood lactate reference parameters. Sixteen boys and nine girls volunteered to participate in the study. They were first tested using a stepwise incremental treadmill protocol to establish the blood lactate profile and peak oxygen uptake ([Vdot]O2). Running speeds corresponding to fixed whole blood lactate concentrations of 2.0, 2.5 and 4.0?mmol?·?l?1 were calculated using linear interpolation. The maximal lactate steady state was determined from four separate 20-min constant-speed treadmill runs. The maximal lactate steady state was defined as the fastest running speed, to the nearest 0.5?km?·?h?1, where the change in blood lactate concentration between 10 and 20?min was?<0.5?mmol?·?l?1. Although the boys had to run faster than the girls to elicit the maximal lactate steady state (15.7 vs 14.3?km?·?h?1, P?<0.01), once the data were expressed relative to percent peak [Vdot]O2 (85 and 85%, respectively) and percent peak heart rate (92 and 94%, respectively), there were no differences between the sexes (P?>0.05). The running speed and percent peak [Vdot]O2 at the maximal lactate steady state were not different to those corresponding to the fixed blood lactate concentrations of 2.0 and 2.5?mmol?·?l?1 (P?>0.05), but were both lower than those at the 4.0?mmol?·?l?1 concentration (P?<0.05). In conclusion, the maximal lactate steady state corresponded to a similar relative exercise intensity as that reported in adult athletes. The running speed, percent peak [Vdot]O2 and percent peak heart rate at the maximal lactate steady state are approximated by the fixed blood lactate concentration of 2.5?mmol?·?l?1 measured during an incremental treadmill test in boys and girls.  相似文献   

16.
This study examined differences in children’s body mass index (BMI) and body fatness (BF%) as a function of gender and fundamental movement skill (FMS) proficiency. Following ethics approval and parental consent, 248, 6–11 year-old children (112 boys, 136 girls) underwent assessment of 7 FMS: sprint run, side gallop, hop, kick, catch, throw and vertical jump. FMS tertiles (“high”, “medium” or “low” FMS) were created based on the summed components of the FMS. Skinfold measures were used to calculate BF%. Physical activity (PA) was assessed using pedometry and maturation predicted using anthropometry. Data were analysed using a 2 (Gender) × 3 (FMS tertile) ways analysis of covariance (ANCOVA), controlling for age, maturation and PA. Age (P = .001) and maturation (P = .006) were associated with BMI. Girls classified as high FMS proficiency had significantly lower BMI compared to girls with low and medium FMS proficiency. Age (P = .0001) and maturation (P = .007) were associated with BF%. BF% was also higher for girls with low FMS compared to those with medium and high FMS. BF% and BMI were not different across FMS tertile in boys. Such findings suggest focusing on FMS may be especially important for healthy weight, particularly in girls.  相似文献   

17.
The purpose of this study was to investigate the acute effect of cigarette smoking on cardiac autonomic function in young adult smokers during dynamic exercise. Fourteen healthy young smokers (21.4 ± 3.4 years) performed peak and submaximal exercise protocols under control and smoking conditions. Resting and submaximal beat-to-beat R-R series were recorded and spectrally decomposed using the fast Fourier transformation. Smoking resulted in a significant decrease in work time, VO(2peak) and peak O(2) pulse (P < 0.05). Heart rate increased at rest and during submaximal exercise after smoking (P < 0.05). The raw high frequency and low frequency power were significantly reduced by smoking, both at rest and during exercise (P < 0.05). The low to high frequency ratio was higher after smoking (P < 0.05). The normalised low frequency power was also significantly increased by smoking, but only at rest (P < 0.05). These data demonstrate that the tachycardic effect elicited by smoking is accompanied by acute changes in heart rate spectral components both at rest and during exercise. Therefore, the cardiac autonomic control is altered by smoking not only at rest, but also during exercise, resulting in reduced vagal modulation and increased sympathetic dominance.  相似文献   

18.
The aim of this study was to objectively quantify ratings of perceived enjoyment using the Physical Activity Enjoyment Scale following high-intensity interval running versus moderate-intensity continuous running. Eight recreationally active men performed two running protocols consisting of high-intensity interval running (6 × 3 min at 90% VO(2max) interspersed with 6 × 3 min active recovery at 50% VO(2max) with a 7-min warm-up and cool down at 70% VO(2max)) or 50 min moderate-intensity continuous running at 70% VO(2max). Ratings of perceived enjoyment after exercise were higher (P < 0.05) following interval running compared with continuous running (88 ± 6 vs. 61 ± 12) despite higher (P < 0.05) ratings of perceived exertion (14 ± 1 vs. 13 ± 1). There was no difference (P < 0.05) in average heart rate (88 ± 3 vs. 87 ± 3% maximum heart rate), average VO(2) (71 ± 6 vs. 73 ± 4%VO(2max)), total VO(2) (162 ± 16 vs. 166 ± 27 L) or energy expenditure (811 ± 83 vs. 832 ± 136 kcal) between protocols. The greater enjoyment associated with high-intensity interval running may be relevant for improving exercise adherence, since running is a low-cost exercise intervention requiring no exercise equipment and similar relative exercise intensities have previously induced health benefits in patient populations.  相似文献   

19.
The purpose of the present study was to re-examine the relationship between deep body temperature and relative exercise intensity, during running rather than cycling (Saltin and Hermansen, 1966). Twenty male competitive and recreational distance runners, aged 22 + 0.9 years (mean +/- sx), were selected to form two groups, one with high maximal oxygen uptake (VO2max) values (72.8 +/- 0.8 ml x kg(-1) x min(-1)) and the other with moderate values (59.4 +/- 0.7 ml x kg(-1) x min(-1)). The participants completed two 60 min constant-paced treadmill runs at a common speed (absolute intensity) of 10.5 km x h(-1) and at a relative exercise intensity at a speed equivalent to 65% of VO2max. During the relative exercise intensity trial, no differences were found in rectal temperature, skin temperature or heart rate between groups. However, when running at the common speed, differences were identified in rectal temperature. At 60 min, rectal temperature was 37.70 +/- 0.19 degrees C and 38.19 +/- 0.11 degrees C for the high and moderate VO2max groups, respectively (P < 0.05). Sweat lost was significantly higher in the moderate VO2max group (moderate: 1.05 +/- 0.06 kg x h(-1); high: 0.82 +/- 0.08 kg x h(-1); P < 0.05). Heart rates were also different between groups over the first 20 min during the common speed trial (P < 0.05). The results of the present study support the findings of Saltin and Hermansen (1966), in that the set-point at which temperature is maintained is related to the relative exercise intensity.  相似文献   

20.
Twenty specialist marathon runners and 23 specialist ultra-marathon runners underwent maximal exercise testing to determine the relative value of maximum oxygen consumption (VO2max), peak treadmill running velocity, running velocity at the lactate turnpoint, VO2 at 16 km h-1, % VO2max at 16 km h-1, and running time in other races, for predicting performance in races of 10-90 km. Race time at 10 or 21.1 km was the best predictor of performance at 42.2 km in specialist marathon runners and at 42.2 and 90 km in specialist ultra-marathon runners (r = 0.91-0.97). Peak treadmill running velocity was the best laboratory-measured predictor of performance (r = -0.88(-)-0.94) at all distances in ultra-marathon specialists and at all distances except 42.2 km in marathon specialists. Other predictive variables were running velocity at the lactate turnpoint (r = -0.80(-)-0.92); % VO2max at 16 km h-1 (r = 0.76-0.90) and VO2max (r = 0.55(-)-0.86). Peak blood lactate concentrations (r = 0.68-0.71) and VO2 at 16 km h-1 (r = 0.10-0.61) were less good predictors. These data indicate: (i) that in groups of trained long distance runners, the physiological factors that determine success in races of 10-90 km are the same; thus there may not be variables that predict success uniquely in either 10 km, marathon or ultra-marathon runners, and (ii) that peak treadmill running velocity is at least as good a predictor of running performance as is the lactate turnpoint. Factors that determine the peak treadmill running velocity are not known but are not likely to be related to maximum rates of muscle oxygen utilization.  相似文献   

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