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本文观察了两种不同方式的力量练习对血清CK、Mb和尿3-MH水平及肌肉酸痛的影响。结果显示：第二次练习的负荷递增方式为“阶梯式”,其练习后血清CK、Mb水平和尿3－MH排泄量的升高幅度明显低于第一次“斜坡式”练习后,且恢复较快,肌肉酸痛程度也有所降低。提示：适宜的训练方式可能减轻由于高强度力量训练引起的骨骼肌纤维损伤。 相似文献
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运动的强度和时间对运动后血清磷酸肌酸激酶和乳酸脱氢酶活性的影响 总被引：2，自引：0，他引：2
本文对51名健康男子不同强度和时间运功前后血清磷酸肌酸激酶(CPK)和乳酸脱氮晦(LDH)活性进行测定和探讨。结果表明,较大强度和较短时间运动后血清CPK和LDH活性变化显著地大于较长时间和较小强度的运动,无训练者运动后CPK活性变化明显地大于有训练者,运动后血清CPK变化的高峰时间表现为, 强度大较强度小出现的晚,无训练者较有训练者出现的晚。 相似文献
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钱光鉴 《上海体育学院学报》1983,(3)
举重运动员在大运动量训练中,会不断地出现局部肌肉酸痛,乃至受伤。如三大力量(上拉、腰力和腿力)训练,随着运动量不断增加,强度逐渐增大,肌肉反应十分明显。有时酸痛持续一周之久,给计划的完成和训练的系统性带来一定的影响。这是当前大运动量训练中亟待解决的问题。肌肉受伤,原因繁多。而总的可归结为两种:一种是在强力作用下肌肉组织突然撕裂或超越肌肉牵伸范围而拉伤;另一种是由肌肉酸痛开始,导致肌肉过度疲劳而受伤。第一种在有关资料中已有详细的 相似文献
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大蒜素与联合抗氧化剂干预对运动员DOMS和CK-MM、CK等的影响 总被引：1，自引：0，他引：1
目的:探讨大蒜素与联合抗氧化剂干预对运动员大强度离心运动后不同时相肌肉酸痛程度、血CK-MM、CK、LDH活性的影响。方法:运动员24名,随机均衡分为实验对照组(A组)、联合抗氧化剂干预组(B组)和大蒜素干预组(C组),运动前2周至运动后2天每天分别口服安慰剂、联合抗氧化剂和大蒜素肠溶胶丸。服药两周后进行一次大强度离心跑台运动,建立运动员运动延迟性肌肉酸痛(DOMS)模型,在服药前、运动前和运动后即刻、运动后24h、运动后48h分别抽取各组运动员肘正中静脉血,测定CK-MM、CK、LDH活性及运动后相应时相肌肉酸痛程度。结果:三组运动员运动后肌肉酸痛程度均呈递增趋势,B、C组运动员运动后各时相肌肉酸痛程度与血CK-MM、CK、LDH活性均低于A组同时相。结论:大强度离心跑台运动可诱发运动员DOMS。大蒜素和联合抗氧化剂可减少运动员大强度离心运动后骨骼肌细胞内CK-MM、CK、LDH的大量逸出,对保护细胞膜完整性、降低运动应激程度、防治运动性骨骼肌微损伤有积极作用,可有效减轻运动员大强度运动后DOMS程度。 相似文献
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通过对1984年2月至2015年2月中国知网和PubMed数据库评定延迟性肌肉酸痛的生理与生化指标进行系统评述,寻找延迟性肌肉酸痛检测指标出现时间以及消失时间,为延迟性肌肉酸痛的预防及治疗提供可靠的理论依据。结果表明在生化指标中血清肌酸激酶（ CK）、乳酸脱氢酶（ LDH）与血清肌红蛋白（ Mb）这三种指标对评定延迟性肌肉酸痛的出现时间、持续时间、不同时间的变化程度等较为灵敏且持续时间较长;在生理指标中细胞内钙离子的活度、巨噬细胞的副产物和肌肉的疼痛特征这三种指标对评定延迟性肌肉酸痛的出现原因及位置、运动后出现的时间及变化程度、持续时间等较其它指标更为明显且容易观测。 相似文献
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《体育科技文献通报》1997,(11)
G884.014.7 9704890大强度力量训练后不同水平举重运动员血清肌酸激酶的变化[刊,中,A]/张莉,周卫海,姚俊//广州体育学院学报.-1997.-17(2).-26-29表2(DW)举重//大强度//血清酶//肌酸激酶//CK//活性通过对不同水平举重运动员大强度力量练习后,血清肌酸激酶(CK)水平差异的观察,表明大强度运动后,高水平运动员和一般运动员的CK 活性均明显升高,但高水平运动员的 CK 活性明显低于一般运动员,且差别有显著意义. 相似文献
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肌肉酸痛是体育运动中经常发生的,长期得不到重视会引发各种综合症。运动后造成的肌肉酸痛原因包括:运动过程中的乳酸堆积;剧烈运动时的肌肉缺氧;肌肉强烈收缩产生的损伤;运动后的肌肉痉挛导致血液循环较差。师范专科学校女生比例很大,体育教学、训练后的肌肉酸痛尤其普遍,曾经有针对性地做过对比试验,试验表明,伸展练习对于缓解局部肌肉的酸痛有一定的积极作用。 相似文献
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大强度游泳抑制小鼠骨骼肌谷氨酰胺合成酶活性 总被引：9，自引：0，他引：9
分析不同强度运动负荷对血浆和骨骼肌谷氨酰胺水平变化与骨骼肌谷氨酰胺合成酶活性变化，探讨运动影响血浆谷氨酰胺水平变化的机制。BAL/C小鼠不负重或2%负重不同强度游泳，每天2h，持续8周。与对照组比较，大强度游泳组动物血浆（0.46±0.10、0.12±0.02mmol/L，p<0.01）和肌肉（4.92±0.98、1.03±0.37mmol/L，p<0.01）中的谷氨酰胺浓度降低，骨骼肌谷氨酰胺合成酶活性抑制(1874±191、1246±220nmol/min/g蛋白,p<0.01)；而中等强度游泳升高血浆谷氨酰胺浓度(0.64±0.06mmol/L,p<0.01)。小鼠在中等强度运动时，谷氨酰胺代谢表现了积极的代偿变化。但在大强度运动时，动物的体重增长减缓，血浆和肌肉谷氨酰胺水平均大幅度降低。结果提示，导致血浆谷氨酰胺水平降低的机制之一是骨骼肌中的谷氨酰胺合成酶抑制。 相似文献
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Sunita Mathur A. William Sheel Jeremy D. Road W. Darlene Reid 《Cardiopulmonary Physical Therapy Journal》2010,21(1):5-12
Purpose: Skeletal muscle damage occurs following high-intensity or unaccustomed exercise; however, it is difficult to monitor damage to the respiratory muscles, particularly in humans. The aim of this study was to use clinical measures to investigate the presence of skeletal muscle damage in the inspiratory muscles. Methods: Ten healthy subjects underwent 60 minutes of voluntary inspiratory threshold loading (ITL) at 70% of maximal inspiratory pressure. Maximal inspiratory and expiratory mouth pressures, delayed onset muscle soreness on a visual analogue scale and plasma creatine kinase were measured prior to ITL, and at repeated time points after ITL (4, 24 and 48 hours post-ITL). Results: Delayed onset muscle soreness was present in all subjects 24 hours following ITL (intensity = 22 ± 6 mm; significantly higher than baseline p = 0.02). Muscle soreness was reported primarily in the anterior neck region, and was correlated to the amount of work done by the inspiratory muscles during ITL (r = 0.72, p = 0.02). However, no significant change was observed in maximal inspiratory or expiratory pressures or creatine kinase. Conclusions: These findings suggest that an intense bout of ITL results in muscle soreness primarily in the accessory muscles of inspiration, however, may be insufficient to cause significant muscle damage in healthy adults.Key Words: delayed onset muscle soreness, respiratory muscles, skeletal muscle damage 相似文献
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大强度训练对散打运动员心血管ET、CGRP、ANP水平的影响 总被引：1，自引：0，他引：1
赵学军 《西安体育学院学报》2003,20(4):57-60
采用放射免疫方法测定9名优秀散打运动员安静时血浆心血管激素,即内皮素(endothelin,ET),降钙素基因相关肽(calcitoningenerelatedpeptide,CGRP)和心钠素(atrialnatriureticpeptide,ANP)水平。经3个月的大强度训练,血浆ET水平逐渐升高,到最后两次取样时的变化明显,提示血浆儿茶酚胺、血管紧张肽分泌增加和(或)骨骼肌和心肌损伤可导致该激素水平提高;自训练开始,血浆CGRP水平依次降低,随着训练的深入其变化明显,表明长时期训练的效果异于急性运动;在大强度训练的进程中ET/CGRP比值逐渐升高,说明运动造成两种激素分泌不匹配;血浆ANP随着训练的深入逐渐升高,并最后两次取样变化明显,说明该激素对训练比较敏感;另外ET和CGRP、CGRP和ANP变化之间有明显的负相关,ET和ANP之间存在明显的正相关;研究显示血液指标的变化与血液浓缩无关。 相似文献
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梁红秀 《山东体育学院学报》2005,21(4):66-67,71
观察大强度运动对大鼠血浆嘌呤核苷酸代谢的影响.大鼠进行大强度运动,运动后血浆尿囊素、次黄嘌呤和肌苷的浓度均比运动前的浓度显著升高（P＜0.01）.提示大强度运动导致大鼠骨骼肌中的腺嘌呤核苷酸丢失. 相似文献
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迷迭香对运动大鼠心肌组织脂质过氧化损伤保护作用的研究 总被引：5，自引：0，他引：5
选用SD大鼠，递增强度训练建立运动模型，研究迷迭香对运动大鼠心肌脂质过氧化损伤的保护作用。结果显示：迷迭香可显著降低血清谷氨酸草酰乙酸转氨酶（sGOT）的活性（P〈0．05），在心肌组织中谷氨酸草酰乙酸转氨酶的活性液呈现下降趋势；迷迭香可以不同程度地增强心肌组织中抗氧化酶SOD、CAT和GSH-Px的活性，均有显著性差异（P〈0．05）；迷迭香可以降低心肌组织中MDA的含量，有显著性差异（P〈0．05）。结论：迷迭香可以增强心肌组织中的抗氧化酶活性，减轻大强度耐力训练对大鼠心肌组织造成的脂质过氧化损伤。 相似文献
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不同负荷训练对大鼠骨骼肌细胞膜自由基代谢、膜流动性及Na~+-K~+-ATP酶活性的影响 总被引：4，自引：0，他引：4
探讨不同负荷游泳训练对大鼠骨骼肌细胞膜结构和功能的影响。分别测试不同负荷下膜的抗氧化酶SOD、膜脂质过氧化产物MDA、膜荧光偏振度(P)及膜Na+-K+-ATP酶活性的变化。与对照组相比,小负荷训练组骨骼肌细胞膜P值降低,膜SOD活性、Na+-K+-ATP酶活性提高,膜上MDA含量则降低;大负荷训练组较小负荷组肌膜P值增加,膜SOD活性、Na+-K+-ATP酶活性下降。适宜负荷训练可以提高骨骼肌细胞膜的抗氧化能力,改善膜脂的流动性,使膜Na+-K+-ATP酶活性增强,而过大负荷训练则使骨骼肌细胞膜的抗氧化能力、膜脂的流动性及膜酶活性降低。 相似文献
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Anastasia G Beneka Paraskevi K Malliou Victoria Missailidou Athanasios Chatzinikolaou Ioannis Fatouros Vassilios Gourgoulis 《Journal of sports sciences》2013,31(3):335-343
Abstract To determine the time course of performance responses after an acute bout of plyometric exercise combined with high and low intensity weight training, a 3-group (including a control group), repeated-measures design was employed. Changes in performance were monitored through jumping ability by measuring countermovement and squat jumping, and strength performance assessment through isometric and isokinetic testing of knee extensors (at two different velocities). Participants in both experimental groups performed a plyometric protocol consisting of 50 jumps over 50 cm hurdles and 50 drop jumps from a 50 cm plyometric box. Additionally, each group performed two basic weight exercises consisting of leg presses and leg extensions at 90–95% of maximum muscle strength for the high intensity group and 60% of maximum muscle strength for the low intensity group. The results of the study suggest that an acute bout of intense plyometric exercise combined with weight exercise induces time-dependent changes in performance, which are also dependent on the nature of exercise protocol and testing procedures. In conclusion, acute plyometric exercise with weight exercise may induce a substantial decline in jumping performance for as long as 72 hours but not in other forms of muscle strength. 相似文献
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The purpose of this study was to evaluate changes in muscle soreness and serum enzyme activity following consecutive drop jumps. Seven male subjects (mean age 30.6 years) performed drop jumps from a 80-cm box height every 7 s until exhaustion (mean = 114 drop jumps). A questionnaire was used to assess muscle soreness (0 = no pain, 7 = unbearable painful) both pre- and post-exercise (0, 12, 24, 36 and 48 h, and 3, 4 and 5 days after the exercise). Blood samples were also taken from three subjects at each of these times. For the other four subjects, blood samples were taken pre-exercise and 0, 12 and 36 h and 5 days post-exercise only. Although there was large inter-subject variability in the development of muscle soreness, all the subjects reported muscle soreness in their lower extremity muscles, especially in the quadriceps femoris. Muscle soreness developed significantly (P less than 0.01) over time, its peak (mean +/- S.E. = 3.7 +/- 0.7) occurring 12-48 h post-exercise. Serum enzyme activity changed significantly over time (P less than 0.05), but the changes were small. Not one subject showed a large increase in creatine kinase, and the average increase was less than 1.3 times as much as the pre-exercise level throughout the period of study. These results suggest that the muscle damage that occurs after drop jumping is not associated with a large release of muscle enzymes into the blood, and muscle soreness is not necessarily related to enzyme elevation following drop jumps. 相似文献
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《European Journal of Sport Science》2013,13(4):337-344
AbstractExercise-induced muscle damage (EIMD), described as the acute weakness of the musculature after unaccustomed eccentric exercise, increases oxidative metabolism at rest and during endurance exercise. However, it is not known whether oxygen uptake during recovery from endurance exercise is increased when experiencing symptoms of EIMD. Therefore, the purpose of this study was to investigate the effects of EIMD on physiological and metabolic responses before, during and after sub-maximal running. After a 12 h fast, eight healthy male participants completed baseline measurements comprising resting metabolic rate (RMR), indirect markers of EIMD, 10 min of sub-maximal running and 30 min of recovery to ascertain excess post-exercise oxygen consumption (EPOC). Measurements were then repeated at 24 and 48 h after 100 Smith-machine squats. Data analysis revealed significant (P<0.05) increases in muscle soreness and creatine kinase (CK) and decreases in peak knee extensor torque at 24 and 48 h after squatting exercise. Moreover, RMR, physiological, metabolic and perceptual responses during sub-maximal running and EPOC were increased in the two days after squatting exercise (P<0.05). It is suggested that the elevated RMR was a consequence of a raised energy requirement for the degradation and resynthesis of damaged muscle fibres. The increased oxygen demand during sub-maximal running after muscle damage was responsible for the increase in EPOC. Individuals engaging in unaccustomed resistance exercise that results in muscle damage should be mindful of the increases in resting energy expenditure and increased metabolic demand to exercise in the days that follow. 相似文献